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Many of the phenotypes introduced in Fig. two ended up examined in the context of protease-activated receptor activation by thrombin or PAR4-AP. While our info unveiled altered outside-in signaling in unstimulated platelets, a number of experiments have offered proof for agonist-induced enhancement of FAK and ERK signaling in platelets and https://pro33slotgacor83603.bloguerosa.com/25883182/top-latest-five-pro33-rtp-gacor-urban-news

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